Ongoing research at Arasys Perfector LLC is investigating the enhancement of endogenous production of Free T3 and IGF-1 via an electronically designed ionic signal, invented by G. Pollock in 2008 after 20 years of research, in Innovations Science, a European Community-funded research center. Pollock’s bio-identical signal initially targets the motor neurons resulting in rhythmical muscle contractions equivalent to performing high resistance physical activity. The process is initiated at the peripheral motor neuron, then the circuit is completed by outgoing CNS neuron emission. This CNS emission causes the ultimate production of Free T3 and GH/IGF-1, which in turn cause lipolysis and muscular hypertrophy. The enhanced production of Free T3 and GF/IGF-I will temporarily cause hyperglycemia. However, the hyperglycemia will resolve once the glucose has been utilized for metabolic purposes including increased cellular energy and muscular hypertrophy.
Blood sample testing reveals that after a series of twenty minute treatments which is accompanied by a subjective sense of enhanced wellbeing in some of the subjects involved in this pilot study.
Post treatment blood samples show healthy blood cells which are uniform in size, color, and shape and float freely in their own space; unlike unhealthy blood cells that are stuck together, a condition that compromises their ability to transport oxygen, nutrients, antibodies or hormones. Therefore, ionic currents separate red blood cells enhancing the transport of oxygen, nutrients, antibodies, hormones to sites of action, as well as waste products to kidneys.
The main focus of the study was to show that ionic current enhance the transport of hormones, as well as increase the secretion of endogenous hormones, specifically T4, T3, GH and IGF-1. A number of studies have shown that thyroid concentrations and serum Free T3 and T4 concentrations are significantly lower in obese non-insulin-dependent diabetics than control subjects. Low T3 is also a strong predictor of mortality in cardiac patients and may be directly implicated in the poor prognosis of cardiac patients.